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Clonal evolution of monosomy 7 in acquired severe aplastic anemia: Two cases treated with allogeneic hematopoietic stem cell transplantation
Figure 1. The first bone marrow biopsy of case 1 (2001) diagnosed as aplastic anemia. It was hypocellular without any hematopoietic elements (a,b). The aspiration smear revealed lymphocytes and plasma cells (c,d).
Clonal evolution of monosomy 7 in acquired severe aplastic anemia: Two cases treated with allogeneic hematopoietic stem cell transplantation
Figure 2. The bone marrow biopsy of case 1 in 2005 was diagnosed as RAEB-II. The biopsy was hypercellular (a) with increased immature erythroid precursors (b,c) and CD34 positive blasts (c,d).
Clonal evolution of monosomy 7 in acquired severe aplastic anemia: Two cases treated with allogeneic hematopoietic stem cell transplantation
Figure 3. FISH analysis of monosomy 7. One probe (orange) was used to evaluate the band7q31 (LSI D7S486) region of chromosome 7 and the other probe (green) was used to evaluate the 7p11.1-q11.1 locus D7ZI region at the centromere. In normal interphase nucleus, two signals from each probe are obtained. However, in presence of monosomy 7, only one signal from each probe is obtained.
T-helper-1 (Th1) and Th2 cytokines after allogeneic hematopoietic stem cell transplantation (allo-HSCT)
Figure 1. TNF-? and IL-2 expression at S1 according to acute GvHD grade (p=0.04 and p=0.08, respectively)
Diffuse alveolar hemorrhage associated with thrombotic thrombocytopenic purpura after allogeneic bone marrow transplantation
Figure 1. Serum LDH, platelet, hemoglobin and creatinine levels of patient. With the increasing LDH levels, platelet and hemoglobin levels showed decrements and microangiopathic hemolytic anemia/TTP occurred. *On day +23, serum creatinine level increased from 1.2 mg/dL to 2.1 mg/dL with a blood CsA level of 500 ng/mL and CsA was ceased. **On day +37, acute GVHD grade II was diagnosed. ***On day +39, despite the regression of acute GVHD, BMT associated TTP was noted. Cyclosporine A was ceased again and plasmapheresis was started.
Diffuse alveolar hemorrhage associated with thrombotic thrombocytopenic purpura after allogeneic bone marrow transplantation
Figure 2a. Posteroanterior chest X ray film showing bilateral diffuse alveolar opacities.
Diffuse alveolar hemorrhage associated with thrombotic thrombocytopenic purpura after allogeneic bone marrow transplantation
Figure 2b. Thorax computed tomogram of the chest showing diffuse alveolar hemorrhage.
Hematopoetic stem celi transplantation activity at a single center: Cerrahpaşa experience
Figure 1. Overall survival of patients with leukenia after allogeneic STC according to diagnosis ( Lines represent: CML -- ,ALL and AML).Cumulative proportion of the patients surviving after transplant. Survival was calculated in months after transplant. Each cross represent a patient being alive at that time.Difference in OS between patients with CML and ALL/AML was highly significant(p
Hematopoetic stem celi transplantation activity at a single center: Cerrahpaşa experience
Figure 2. Overall survival of patients with leukenia after allogeneic SCT according to disease status at transplant (Lines represent: Early-stage--- and advanced-stage).Cumulative proportion of the patients surviving after transplant.Survival was calculated in months after transplant. Each cross represent a patient being alive at that time.Difference in OS between patients transplanted in early stages and advanced stages was highly significant(p
Hematopoetic stem celi transplantation activity at a single center: Cerrahpaşa experience
Figure 3. Disease-free survival after allogeneic SCT according to diagnosis(Lines represent: CML----,ALL and AML).Cumulative proportion of the patients surviving after transplant.Survival was calculated in months after CR was documented. Each cross represent a patient being alive at the time.Difference in DFS between patients with CML and ALL/AML was highly significant(p
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